Who Ya Gonna Call Before August 16 to Complain about 4G and 5G Small Cell Towers Being Installed in Front of Homes, etc.? The Senate Commerce, Science and Transportation Committee



Who Ya Gonna Call Before August 16 to Complain about 4G and 5G Small Cell Towers Being Installed in Front of Homes, etc.? The Senate Commerce, Science and Transportation Committee (Contact Info Provided).

https://www.activistpost.com/2018/08/call-before-august-16-complain-4g-5g-towers-contact-list.html

August 8, 2018

By B.N. Frank

Big Thanks to all EMF activists for providing so much information to Activist Post so we can pass it on to readers who also want to stop the widespread “Roll Out” of risky 5G technology and other Telecom Industry, FCC, and other elected officials’ nonsense.

Since 2004, The International Association of Firefighters has opposed the use of their stations as base stations for cell towers and antennas until it can be proven that this is NOT hazardous to their health. So why would any of the rest of us be okay with allowing small cell towers to be installed in front of our homes, in public rights-of-ways, and everywhere else? This violates “The Precautionary Principle.”

In addition to contacting your local, state, and federal elected officials ASAP, a list of senate committee members and their contact information is posted at the end of this article. Please contact them before August 16.

With a little luck, we can help it out. We can make this whole damn thing work out.

Here’s the deal as relayed via excerpts from an article posted by Radio + TV Business Report:

On Aug, 16 the Federal Communications Commission (FCC) has an oversight hearing which will be conducted by Senate Commerce, Science and Transportation Committee.

According to Committee Chairman John Thune (R-S.D.)

The hearing, the Committee notes, will examine policy issues before the Commission and review the FCC’s ongoing duties and activities. This includes efforts to better utilize spectrum powering our wireless economy to expanding rural broadband access, combatting robocalls, and reviewing the media landscape.

(A little more background):

For several hours on July 25, the four voting members of the FCC answered questions and, in a handful of instances, sparred with House of Representatives members who wanted assurances that the White House would not sway the Commission on its decision to send Sinclair Broadcast Group‘s intended merger with Tribune Media to an Administrative Law Judge.

Because of this, the FCC Four went to the Senate side of Capitol Hill for an oversight hearing that was conducted by Senate Commerce, Science and Transportation Committee one day later than originally planned.

On July 30, the committee announced that it would convene its hearing on August 15 at 10:15am. However, now it’s been pushed back again and will be held on August 16 at 10am at Russell Senate Office Building, Room 253.

Witness testimony, opening statements, and a live video of the hearing will be available on http://www.commerce.senate.gov.

The following list of committee members and their contact information was provided by EMF activists. Websites are not working for some officials and this has been noted next to their names. Please contact some or all of them before August 16 and let them know how you feel about all of this:

Committee on Commerce, Science, and Transportation

https://www.commerce.senate.gov/public/index.cfm/committeemembers

Contact Information for the Committee Full Committee Office
Majority: 202-224-1251
Majority Address: 512 Dirksen Senate Building; Washington DC, 20510
Minority: 202-224-0411

MAJORITY MEMBERS:

1. CHAIRMAN: Senator John Thune, South Dakota
Washington D.C. Office
United States Senate SD-511
Washington, DC 20510
Phone: (202) 224-2321
Fax: (202) 228-5429
Toll-Free: 1-866-850-3855
EMAIL FORM: https://www.thune.senate.gov/public/index.cfm/contact

2. Senator Roger Wicker Mississippi
Washington, D.C.
555 Dirksen Senate Office Building
Washington, DC 20510
Main: (202) 224-6253
Fax: (202) 228-0378
EMAIL FORM https://www.wicker.senate.gov/public/index.cfm/contact

3. Senator Roy Blunt Missouri
Washington, D.C.
260 Russell Senate Office Building
Washington, DC 20510
Phone: (202) 224-5721
EMAIL CONTACT FORM: https://www.blunt.senate.gov/public/index.cfm/contact-roy

4. Senator Ted Cruz Texas
WASHINGTON, D.C.
(202) 224-5922
404 Russell
Washington, DC 20510
EMAIL: https://www.cruz.senate.gov/?p=form&id=16

5. Senator Deb Fischer Nebraska
Washington D.C.
454 Russell Senate Office Building
Washington, DC 20510
Phone: (202) 224-6551
Fax: (202) 228-1325
EMAIL: https://www.fischer.senate.gov/public/?p=email-deb

6. Senator Jerry Moran Kansas
Washington, D.C.
Dirksen Senate Office Building
Room 521
Washington, D.C. 20510
Phone: (202) 224-6521
Fax: (202) 228-6966
https://www.moran.senate.gov/public/index.cfm/e-mail-jerry

7. Senator Dan Sullivan Alaska
WASHINGTON, D.C.
702 Hart Senate Office Building
Washington, DC 20510
Phone: (202)-224-3004
Fax: (202)-224-6501
https://www.sullivan.senate.gov/contact/email

8. Senator Dean Heller Nevada
Washington, DC
324 Hart Senate Office Building
Washington, DC 20510
Phone: 202-224-6244
Fax: 202-228-6753
https://www.heller.senate.gov/public/index.cfm/contact-form

9. Senator Jim Inhofe Oklahoma (site down)

10. Senator Mike Lee Utah (site down)

11. Senator Ron Johnson Wisconsin
328 Hart Senate Office Building
Washington, DC 20510
Phone: (202) 224-5323
Fax: (202) 228-6965
https://www.ronjohnson.senate.gov/public/index.cfm/email-the-senator

12. Senator Shelley Moore Capito West Virginia

13. Senator Cory Gardner Colorado
Washington, D.C.
354 Russell
Senate Office Building
Washington, DC 20510
P: (202) 224-5941
F: (202) 224-6524
https://www.gardner.senate.gov/contact-cory/email-cory

14. Senator Todd Young Indiana (site down)

MINORITY MEMBERS:

1. Ranking Member Bill Nelson Florida
WASHINGTON, DC OFFICE
United States Senate
716 Senate Hart Office Building
Washington, DC 20510
Phone: 202-224-5274
Fax: 202-228-2183
https://www.billnelson.senate.gov/contact-bill

2. Senator Maria Cantwell Washington
Washington, DC
511 Hart Senate Office Building
Washington, DC 20510
Phone: (202) 224-3441
Fax: (202) 228-0514
https://www.cantwell.senate.gov/contact/email

3. Senator Amy Klobuchar Minnesota
Washington, DC
302 Hart Senate Office Building
Washington, DC 20510
phone: 202-224-3244
fax: 202-228-2186
https://www.klobuchar.senate.gov/public/index.cfm/email-amy

4. Senator Richard Blumenthal Connecticut
https://www.blumenthal.senate.gov/contact
Washington D.C.
706 Hart Senate Office Bldg.
Washington, DC, 20510
tel (202) 224-2823
fax (202) 224-9673

5. Senator Brian Schatz Hawaii
722 HART SENATE OFFICE BUILDING
WASHINGTON, DC 20510
PHONE: (202) 224-3934
FAX: (202) 228-1153
https://www.schatz.senate.gov/contact

6. Senator Ed Markey Massachusetts
Washington, D.C.
255 Dirksen Senate Office Building
Washington, D.C. 20510
202-224-2742
https://www.markey.senate.gov/contact

7. Senator Tom Udall New Mexico
Washington/Capitol Hill
531 Hart Senate Office Building
Washington DC, 20510
(202) 224-6621
https://www.tomudall.senate.gov/contact/email-tom

8. Senator Gary Peters Michigan
Hart Senate Office Building
Suite 724
Washington, DC 20510
(202) 224-6221
https://www.peters.senate.gov/contact/email-gary

9. Senator Tammy Baldwin Wisconsin (site down)

10. Senator Tammy Duckworth Illinois (site down)

11. Senator Maggie Hassan New Hampshire (site down)

12. Senator Catherine Cortez Masto Nevada (site down)

13. Senator Jon Tester Montana (site down)

There is no end to what we can do together. There is no end.

Free ebook How To Survive the Job Automation Apocalypse
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And Here, You Thought Fukushima Had Ended, Silly You!


ENENews.com – Energy News

Report: Massive radiation leak at Fukushima plant — Extremely high levels being detected outside reactor — Officials can’t explain why — Expert warns of global threat: “It’s a disaster of unseen proportions” (VIDEO)
http://enenews.com/report-massive-radiation-leak-at-fukushima-plant-extremely-high-levels-found-outside-reactor-expert-warns-of-global-threat-its-a-disaster-of-unseen-proportions-video
Published: February 6th, 2018 at 7:23 am ET
By ENENews

The Independent, Feb 2, 2018 (emphasis added): Fukushima nuclear disaster: Lethal levels of radiation detected in leak… Expert warns of ‘global’ consequences unless the plant is treated properly… [Tepco] found eight sieverts per hour of radiation, while 42 [sieverts] were also detected outside its foundations… It came as Tepco said the problem of contaminated water pooled around the plants three reactors that is seeping into the ground has caused a major headache in its efforts to decommission the plant… Mycle Schneider, an independent energy consultant and lead author of the World Nuclear Industry Status Report, said that Tepco “hasn’t a clue what it is doing” in its job to decommission the plant. He added that the contaminated water that is leaking at the site could end up in the ocean if the ongoing treatment project fails and cause a “global” disaster, he told The Independent… “I find it symptomatic of the past seven years, in that they don’t know what they’re doing, Tepco, these energy companies haven’t a clue what they’re doing, so to me it’s been going wrong from the beginning. It’s a disaster of unseen proportions.” Mr Schneider added that the radiation leaks coupled with the waste from the plant stored in an “inappropriate” way in tanks could have global consequences… “This can get problematic anytime, if it contaminates the ocean there is no local contamination, the ocean is global, so anything that goes into the ocean goes to everyone.” He added: “It needs to be clear that this problem is not gone, this is not just a local problem. It’s a very major thing.”

NHK, Feb 1, 2018: High radiation detected at Fukushima plant… A remote-controlled inspection of the Unit 2 reactor containment vessel last month detected a maximum of 8 sieverts per hour of radiation… [Tepco] said the radiation reading was taken near what appeared to be fuel debris, the term used to describe a mixture of molten fuel and broken interior parts… radiation levels remain so high that they present a major challenge to decommissioning work. During the probe, 42 sieverts per hour of radiation was also detected outside the foundations of the reactor. But officials said they have doubts about the accuracy of the reading because a cover had not been removed from the measuring instrument at the time. They added that they don’t know why radiation levels were lower near the suspected fuel debris than around the foundations. They gave a number of possible reasons, such as that cooling water may have washed radioactive materials off the debris…

RT, Feb 2, 2018: An inspection of the Fukushima nuclear plant has detected extremely high amounts of radiation, says operator TEPCO… Experts can’t explain why radiation levels in fuel debris were lower than outside the reactor’s foundations…

Sputnik, Feb 4, 2018: ‘Global Consequences’ of Lethal Radiation Leak at Destroyed Japan Nuclear Plant… While 8 Sv/h is deadly, outside of Fukushima’s Reactor Number 2 foundations… a much higher level of 42 Sv/h was detected. A strange occurrence, and experts are still arguing what caused the discrepancy. One possible explanation is that cooling water washed radioactive material off debris, taking it somewhere else. But here’s a truly terrifying catch: according to the report, Tepco highly doubts the new readings, because, as was discovered later, a cover was not removed from the robot-mounted measurement device at the time of the inspection, NHK World reports… While that radiation dosimeter cover negligence prevents precise calculations, the actual picture inside Unit 2 is thought to be much worse…

Watch NHK’s broadcast here:
https://www3.nhk.or.jp/nhkworld/nhknewsline/nuclearwatch/highradiationatfukushimadaiichi7yearson/

Published: February 6th, 2018 at 7:23 am ET

Fukushima is not over, not by a long shot. Every day since March 11, 2011, Japan has allowed the radioactive water to run into the ocean. Japan has threatened their doctors about telling the truth about the illnesses that have plagued the Japanese people since the meltdown, and have arrested and threatened numerous reporters for reporting the truth.
Fukushima has global consequences because all of our oceans connect to one another. Japan is supposed to be readying to release 100 million tons of radioactive water into the Pacific Ocean.

The Donald’s Done — The Deep State Wins Its War On America First; written by david stockman wednesday may 9, 2018


The Donald’s Done — The Deep State Wins Its War On America First
written by david stockman wednesday may 9, 2018
http://www.ronpaulinstitute.org/archives/featured-articles/2018/may/09/the-donalds-done-the-deep-state-wins-its-war-on-america-first/

The Donald’s action to ash-can the Iranian nuclear deal marks the War Party’s complete and baleful triumph. There is now nothing much left of America First.

Trump’s reckless, unwarranted and utterly irrational action will pull Washington ever deeper into an incendiary middle eastern vortex of political and religious conflict that has absolutely nothing to do with the safety and security of the America people.

To the contrary, picking a fight with Tehran is an exercise in unprovoked Imperial aggression. The Iranian regime has no means to attack America militarily and has never threatened to do so. Nor has it invaded any other country in the region where it was not invited by a sovereign government host.

Even Iran’s minor skirmishes with American forces in recent years have been owing to the happenstance of Washington’s far-flung imperial ventures.

For example, Washington destroyed Saddam’s Sunni/secular government in Iraq and installed a Shiite regime in Baghdad, thereby leaving the Sunni lands of western Iraq in chaos. Only then did Baghdad invite their shiite co-religionists from Iran to help excise the scourge of ISIS that formed from the remnants of Saddam’s army and government.

Likewise, Washington and its allies sent thousands of jihadist warriors and billions of aid and supplies into Syria to topple its dully elected government. Only then did the Alawite (Shiite) Assad regime invite help from its confessional compatriots in Tehran.

And you can’t find any more ludicrous example of the cat calling the kettle black than the Donald’s claim that Iran is a terrorist state because it is aligned with the Shiite population of Lebanon represented by Hezbollah.

For crying out loud. The War Party pretends Washington has turned much of the middle east into rubble and barbarism in order to spread democracy — whether they wanted it or not, and whether they were ready for it or not.

But Lebanon is a serviceable democracy and last weekend Hezbollah and its allies — including certain Sunni factions — won an overwhelming election victory. They now control a clear majority in its legislature, where Hezbollah will have the power to name a new Prime Minister (a Sunni) and Speaker of the Parliament (a Shiite) — both of whom will be pledged to work with the country’s Christian president.

That particular outcome of democracy the War Party can’t abide. But it fairly violates the english language itself to call it state sponsored terrorism.

In a similar vein, the Houthi tribe of Shiites have dominated much of northern and western Yemen for centuries. So when a Washington installed government in Sana’a was overthrown, the Houthi took power in northern Yemen — as had been the case during the long expanse from 1918-1990 when the two Yemens were finally unified.


But it is the Houthis who are the victims of aggression by the brutal Saudi bombing campaign that has left more than 10,000 civilians dead and the land plagued with famine, cholera, rubble, and economic collapse.

There is no telling which faction in Yemen’s fratricidal civil war and invasion by Saudi Arabia is the more barbaric, but the modest aid provided by Iran to its Shiite kinsman in northern Yemen is absolutely not a case of state sponsored terrorism.

In a word, the Donald has fallen hook, line and sinker for the War Party’s lie- and propaganda-filled demonization of the Iranian regime. We have debunked this false history elsewhere, but suffice to say that it boils down to two very imperialist propositions.

To wit, that Iran is not entitled to have its own foreign policy via alliances with Iraq, Syria, the dominant party of Lebanon, or the official government in Sana’a Yemen because Washington (and Israel) say so; and that it’s not allowed to have even intermediate and medium range missiles (that can’t reach either the US or most of Europe) to defend itself — even though Washington has armed its far wealthier Sunni rival across the Persian Gulf with upwards of $250 billion of America’s most advanced warplanes, attack helicopters, missiles, drones and sundry other accoutrements of war.

And that is to say nothing of a tiny residual capacity to enrich uranium to 3.5 percent purity (compared to 90 percent weapons grade) for civilian power reactors on fewer than one-fifth of the oldest and slowest centrifuges it had before the 2015 nuke deal.

Nor does it consider that all 17 US intelligence agencies certified in an official NIE (national intelligence estimate) in 2007 and again in 2011 that Iran only had a small weaponization research program between 1999 and 2003, which was then abandoned and never restarted.

Moreover, the documentary proof of that was thoroughly investigated by the IAEA after the 2015 deal, which then re-validated that the Iranian weapons program was indeed disbanded in 2003.

In short, the Donald has fallen for a pack of lies and distortions that are only remotely plausible if the aim is to find enemies and territories around the planet to police, occupy or otherwise hegemonize. And to thereby keep the Warfare State in business, its $800 billion budget funded, and the Imperial City’s vast beehive of think-tanks, contractors, NGOs, lobbyists, and racketeers in clover.

The invincible grip on power of the above — the Deep State for short-hand purposes — has now been proven. And that’s a full-on tragedy because the Donald’s inchoate notion of America First was an incipient challenge to its power — the only one since the end of the cold war.

To be sure, Donald Trump never had a coherent or articulated notion of America First. But all of his impulses were in the right direction.

Perhaps like renegade Sarah Palin before him, for example, he could see Russia from his airy on the 68th floor of Trump Tower and recognize that it is no threat whatsoever to America’s security.

That is, from his perch the Donald could gaze upon metro New York’s $1.6 trillion of GDP, which is greater than the entirety of Russia’s economy ($1.5 trillion GDP); and whether he knew the precise numbers or not, his impulse toward rapprochement with Putin was spot on.

Likewise, whether he had gotten George Bush’s folly in Iraq right on day one or not — he was loud and clear in his consistent denunciation years before Hillary sprouted her dawkish feathers.

Nor was he any less correct when he averred that NATO was obsolete. After all, the GDP of the EU-29 is 10X larger than Russia’s, and their combined military spending is 4X greater.

If you’re not a prisoner of Imperial Washington’s twisted group think you cannot possibly believe that Russia’s supremely rational leader — Cool Hand Vlad — intends to militarily assault his European neighbors. He’d like to supply their markets, not occupy their cities — something that anyone except the demented, self-serving bureaucrats of NATO can easily understand.

Ditto for Afghanistan, Syria, Yemen, Libya, Somalia et. al. They aren’t cold war “dominoes” because the Soviet Union slithered off the pages of history 27 years ago; they don’t threaten America directly, either, because they don’t have two dimes to rub together economically or militarily; and whether they affiliate with the Saudi-Sunni axis or the Iran-Shiite crescent makes not one damn bit of difference for the safety and security of American citizens in Lincoln NE or Springfield MA.

In the case of the Korean Peninsula, the Donald also rightly questioned why we are still funding 29,000 US troops when the war there ended 65 years ago.

The truth is, it is a war that never should have been fought in the first place because the now open Soviet archives show both Stalin and Mao were against it. US national security was never at stake.

Rather than a domino, it originated purely as a civil war between the communist/nationalists under Kim II-sung, who had fought the Japanese occupiers to the death, and a puppet government in the South under Syngman Rhee.

The latter was an aristocratic dandy who moved to the US in 1904 and spent most of the next 40 years hob-knobbing in Washington. So doing, he promoted endless schemes to install himself in power back in Korea, which finally happened when Japan’s 35-year long occupation was ended in August 1945.

At length, the two Korean political rivals got into a war that the north would have handily won — and might well have turned itself into a cheap labor based export platform just as did Mao’s heirs on the mainland. It might even have become a darling of Wall Street — just as the Red Suzerains of Beijing are today.

That is to say, there was exactly nothing at stake in June 1950 — until the rabid cold warriors in Washington persuaded Truman to intervene.

So doing, the US military launched the most destructive and vicious bombing campaign in history under the blood-thirsty top Air Force general, Curtis LeMay. By the 1953 armistice, North Korea had become a bombed-out wasteland with hardly a city or town not reduced to rubble and with millions of civilians dead or starving.

But it was not merely a pointless war and waste of American blood and treasure; it also became forever embraced by the people of the north as the patriotic war of resistance that paved the way for six decades of the brutal Kim family dictatorship and a life of poverty and misery for its 25 million people, who could otherwise be working in Apple factories and auto plants today.

Needless to say, Imperial Washington has no regard for honest history — only its own self-serving narrative and imperative need for enemies and missions to justify nearly $800 billion per year for the machinery of war and empire. In the case of North Korea, in fact, its imperial pretensions and penchant for “regime change” under the neocons in recent decades, unleashed a veritable monster.

That is, a drive by the Kim family to obtain nuclear weapons, thereby hoping to avoid Saddam’s fate at the end of a rope or Khadafy’s bloody demise with a shive up his rectum.

Fortunately, the Donald has been blessed with a historic serendipity. His military bluster and name-calling apparently caused Kim Jong-un to stage so many nuclear bomb tests culminating in a huge (for N. Korea) 160 kiloton explosion last September that the Fat Boy of Pyongyang has literally destroyed the mountains which house his Punggye-ri test site.

A recent authoritative study actually warns that if North Korea were to use the same area for another test it could cause an “environmental catastrophe.”

North Korea’s past tests have altered the tectonic stress in the region to the extent that previously inactive tectonic faults in the region have reached their state of critical failure. Any further disturbance from a future test could generate earthquakes that may be damaging by their own force or crack the nuclear test sites of the past or the present.
Of course, Kim Jong-un is now attempting to make a virtue out of necessity by ostentatiously shuttering the no longer useable site and inviting the world to witness its entombment. But if that leads to a denuclearization of the Korean peninsula, so be it.

And let it also be an occasion to reverse the mistake of June 1950, and get American forces off the peninsula once and for all: Return Korea to the Koreans to work out their own governance as they see fit!

Yet even on this matter, where the Donald has recently tried to explore a drastic reduction, if not complete removal, of US troops as part of the pending deal with Kim Jong-un, the Deep State has come down on him with all fours.

In that regard, here is what former “peace” candidate Barack Obama’s leading advisor on the topic had this to say:

Kelly E. Magsamen, a top Asia policy official at the Pentagon during the Obama administration, said, ‘U.S. presence in South Korea is a sacrosanct part of our alliance.’
In fact, apparently the entire global empire of Washington is sacrosanct — including the ridiculous fact that in the year 2018 Washington still has military bases in the defeated powers of World War II. Yet neither Japan nor Germany have any mortal military enemies and both are utterly dependent on the trade custom of the US for their high standard of living.

So the Deep State now owns the Donald and America First is not even a slogan. It’s inoperative, Nixon-style.

Indeed, it’s only a matter of time before the Donald gets the ultimate Nixon treatment — now that he has done the Deep State’s dirty work and ash-canned the deal that could have opened a broad avenue toward peace in the world and drastic retrenchment of the fiscally bankrupting Warfare State at home.

That is to say, at length the ingrates of the Deep State will put the Donald on the Dick Nixon Memorial Helicopter for his final ride to Gonesville.

To paraphrase the great Randolph Bourne, Demonization of the Unwilling is the Health of the Deep State.

At least that much the Donald has now, regrettably, confirmed with his sophomoric attack on Iran.

So doing he has also lurched America drastically forward on the path to a monumental financial catastrophe. That’s because taken together the Warfare State and the Welfare State are also the fiscal demise of the state.

One of these days even the lemmings of Wall Street — which took day’s calamitous news in stride — will finally get the memo, too.

Reprinted with permission from David Stockman’s Contra Corner.

Sick pelicans showing up along Southern California coast By The Associated Press (Gasp! OMG! How shocking that any are still alive at all. Fukushima is still melting down, like Ddduuuuhhhhh!)



FILE–In this April 28, 2018, file photo, made from video provided by Pepperdine University, shows one of a pair of pelicans crashing a graduation ceremony at Pepperdine University in Malibu, Calif. The wildlife organization, International Bird RescuThe Associated Press

Sick pelicans showing up along Southern California coast
By The Associated Press
LOS ANGELES — May 10, 2018, 5:00 PM ET

FILE–In this April 28, 2018, file photo, made from video provided by Pepperdine University, shows one of a pair of pelicans crashing a graduation ceremony at Pepperdine University in Malibu, Calif. The wildlife organization, International Bird Rescue, said Thursday, May 10, 2018, that there’s been a surge in the number of sick and dying brown pelicans along the Southern California coast in the past week. (Grant Dillion/Pepperdine University via AP, file)
more +

A wildlife organization says there’s been a surge in the number of sick and dying brown pelicans along the Southern California coast in the past week.

International Bird Rescue said Thursday that more than 25 pelicans have been brought to its wildlife center in the San Pedro district of Los Angeles.

The big birds are showing signs of emaciation, hypothermia and anemia. The organization did not cite a cause.

Wildlife center manager Kylie Clatterbuck says it’s normal to receive recently fledged baby pelicans this time of year but the current wave includes many second-year birds.

The organization says there are many cases of pelicans landing on city streets, residential yards and airport runways.

A well-publicized incident occurred April 28 when two pelicans landed at Pepperdine University’s graduation ceremony in Malibu.

———

Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly. In fact, “high LDL-C may be protective is in accordance with the finding that LDL-C is lower than normal in patients with acute myocardial infarction”.



Cardiovascular medicine
Research
Lack of an association or an inverse association between low-density-lipoprotein cholesterol and mortality in the elderly: a systematic review
http://bmjopen.bmj.com/content/6/6/e010401.full?sid=cfb00014-f0a8-407d-ae71-a3278160ca49

Uffe Ravnskov1, David M Diamond2, Rokura Hama3, Tomohito Hamazaki4, Björn Hammarskjöld5, Niamh Hynes6, Malcolm Kendrick7, Peter H Langsjoen8, Aseem Malhotra9, Luca Mascitelli10, Kilmer S McCully11, Yoichi Ogushi12, Harumi Okuyama13, Paul J Rosch14, Tore Schersten15, Sherif Sultan6, Ralf Sundberg16
Author affiliations
Abstract

Objective It is well known that total cholesterol becomes less of a risk factor or not at all for all-cause and cardiovascular (CV) mortality with increasing age, but as little is known as to whether low-density lipoprotein cholesterol (LDL-C), one component of total cholesterol, is associated with mortality in the elderly, we decided to investigate this issue.

Setting, participants and outcome measures We sought PubMed for cohort studies, where LDL-C had been investigated as a risk factor for all-cause and/or CV mortality in individuals ≥60 years from the general population.

Results We identified 19 cohort studies including 30 cohorts with a total of 68 094 elderly people, where all-cause mortality was recorded in 28 cohorts and CV mortality in 9 cohorts. Inverse association between all-cause mortality and LDL-C was seen in 16 cohorts (in 14 with statistical significance) representing 92% of the number of participants, where this association was recorded. In the rest, no association was found. In two cohorts, CV mortality was highest in the lowest LDL-C quartile and with statistical significance; in seven cohorts, no association was found.

Conclusions High LDL-C is inversely associated with mortality in most people over 60 years. This finding is inconsistent with the cholesterol hypothesis (ie, that cholesterol, particularly LDL-C, is inherently atherogenic). Since elderly people with high LDL-C live as long or longer than those with low LDL-C, our analysis provides reason to question the validity of the cholesterol hypothesis. Moreover, our study provides the rationale for a re-evaluation of guidelines recommending pharmacological reduction of LDL-C in the elderly as a component of cardiovascular disease prevention strategies.

This is an Open Access article distributed in accordance with the Creative Commons Attribution Non Commercial (CC BY-NC 4.0) license, which permits others to distribute, remix, adapt, build upon this work non-commercially, and license their derivative works on different terms, provided the original work is properly cited and the use is non-commercial. See: http://creativecommons.org/licenses/by-nc/4.0/

http://dx.doi.org/10.1136/bmjopen-2015-010401
Statistics from Altmetric.com
Article has an altmetric score of 1664

Strengths and limitations of this study

This is the first systematic review of cohort studies where low-density lipoprotein cholesterol (LDL-C) has been analysed as a risk factor for all-cause and/or cardiovascular mortality in elderly people.

Lack of an association or an inverse association between LDL-C and mortality was present in all studies.

We may not have included studies where an evaluation of LDL-C as a risk factor for mortality was performed but where it was not mentioned in the title or in the abstract.

We may have overlooked relevant studies because we have only searched PubMed.

Minor errors may be present because some of the authors may not have adjusted LDL-C by appropriate risk factors.

Some of the participants with high LDL-C may have started statin treatment during the observation period and, in this way, may have added a longer life to the group with high LDL-C and some of them may have started with a diet able to influence the risk of mortality.

We may have overlooked a small number of relevant studies because we only searched papers in English.

Introduction
Rationale

For decades, the mainstream view has been that an elevated level of total cholesterol (TC) is a primary cause of atherosclerosis and cardiovascular disease (CVD). There are several contradictions to this view, however. No study of unselected people has found an association between TC and degree of atherosclerosis.1 Moreover, in most of the Japanese epidemiological studies, high TC is not a risk factor for stroke, and further, there is an inverse association between TC and all-cause mortality, irrespective of age and sex.2

In a recent meta-analysis performed by the Prospective Studies Collaboration, there was an association between TC and CV mortality in all ages and in both sexes.3 However, even in this analysis, the risk decreased with increasing age and became minimal after the age of 80 years. Since atherosclerosis and CVD are mainly diseases of the elderly, the cholesterol hypothesis predicts that the association between CV mortality and TC should be at least as strong in the elderly as in young people. There may be a confounding influence in these studies, however, because TC includes high-density lipoprotein cholestrol (HDL-C), and multiple studies have shown that a high level of HDL-C is associated with a lower risk of CVD.
Objectives

We examined the literature assessing low-density lipoprotein cholesterol (LDL-C) as a risk factor for mortality in elderly people. Since the definition of CVD varies considerably in the scientific literature, we have chosen to focus on the association between LDL-C and all-cause and CVD mortality, because mortality has the least risk of bias among all outcome measures. If Goldstein and Brown’s recent statement that LDL-C is ‘the essential causative agent’ of CVD4 is correct, then we should find that LDL-C is a strong risk factor for mortality in elderly people.


Methods
Search strategy

UR and RS searched PubMed independently from initial to 17 December 2015. The following keywords were used: ‘lipoprotein AND (old OR elderly) AND mortality NOT animal NOT trial’. We also retrieved the references in the publications so as not to miss any relevant studies. The search was limited to studies in English.
Inclusion and exclusion criteria

All included studies should meet the following criteria: the study should be a cohort study of people aged 60 years or older selected randomly from the general population, or a study where the authors had found no significant differences between the participants and the source population’s demographic characteristics. The studies should include an initial assessment of LDL-C levels, the length of the observation time and information about all-cause and/or cardiovascular mortality at the end of follow-up. The studies should also include information about the association between LDL-C and all-cause and/or CVD mortality. We excluded studies that did not represent the general population (eg, case–control studies; case reports; studies that included patients only); studies where data about elderly people were not given separately, and studies without multivariate correction for the association between LDL-C and all-cause and/or CV mortality. We accepted studies where the authors had excluded patients with serious diseases or individuals who had died during the first year.
Study selection, data items and extraction

Studies where the title or abstract indicated that they might include LDL-C data of elderly people, were read in full, and the relevant data were extracted by at least three of the authors, for example, year of publication, total number of participants, sex, length of observation time, exclusion criteria, LDL-C measured at the start and the association between initial LDL-C and risk of all-cause and/or at follow-up. When more than one adjusted HR was reported, the HR with the most fully adjusted model was selected.

Quality assessment

The design of the study satisfies almost all points of reliability and validity according to the Newcastle Ottawa Scale as regards selection, comparability and exposure.5 Thus, all studies represented elderly people only; ascertainness of exposure (eg, measurement of LDL-C) was present in all studies, and outcome was unknown at the start. It can be questioned if all of the studies represented the general population because, as shown below, in some of them various types of disease groups were excluded.

Results
Study selection

Our search gave 2894 hits. We excluded 160 studies, which were not in English, and 2452 studies because, judged from the abstract, it was obvious that they were irrelevant.

The rest of the papers were read in full; 263 of these studies were excluded for the following reasons: (1) the participants did not represent the general population; (2) LDL-C was not measured at the start; (3) follow-up information was not given for the elderly separately; or (4) no information was present about mortality during the observation period (figure 1). One of the studies6 was excluded because it included the same individuals as in a previous study.7
Figure 1

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Figure 1

Flow Chart. CV, cardiovascular; LDL-C, low-density lipoprotein cholesterol.
Study characteristics

The remaining 19 studies including 30 cohorts with a total of 68 094 participants met the inclusion criteria (figure 1). All-cause mortality was recorded in 28 cohorts. In 16 of these cohorts (representing 92% of the individuals), the association was inverse and with statistical significance in 14; in 1 of the cohorts, the association was mirror-J-formed with the lowest risk in the highest quartile; in the rest of the papers, no association was found. CV mortality was recorded in nine cohorts; in one of them, the association was almost U-shaped with the lowest risk in the highest quartile (curvilinear fit: p=0.001); in one of them, the association was mirror-J-formed and also with the lowest risk in the highest quartile (curvilinear fit: p=0.03); in the other seven cohorts, no association was found (table 1).

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Table 1

Association between LDL-C and all-cause mortality and CVD mortality, respectively, in 19 studies including 30 cohorts with 68 094 individuals from the general population above the age of 60 years
Risk of bias across studies

One explanation for the increased risk of mortality among people with low cholesterol is that serious diseases may lower cholesterol soon before death occurs. Evidence to support this hypothesis may be obtained from 10 of the studies in which no exclusions were made for individuals with terminal illnesses. However, in four of the studies, participants with a terminal illness or who had died during the first observation year were excluded. In one of those studies,8 LDL-C was not associated with all-cause mortality; in the three others,16 ,20 ,24 which included more than 70% of the total number of participants in our review, LDL-C was inversely associated with all-cause mortality and with statistical significance. Thus, there is little support for the hypothesis that our analysis is biased by end of life changes in LDL-C levels.

It is also potentially relevant that all studies did not correct for the same risk factors, and some of them did not inform the reader about which risk factors they corrected for. However, taking all studies together, 50 different risk factors were corrected for in the Cox analyses (table 2).

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Table 2

Factors corrected for in the multifactorial analyses of each study

It is worth considering that some of the participants with high LDL-C may have started statin treatment during the observation period. Such treatment may have increased the lifespan for the group with high LDL-C. However, any beneficial effects of statins on mortality would have been minimal because most statin trials have had little effect on CVD and all-cause mortality, with a maximum reduction of mortality of two percentage points. It is therefore relevant that the 4-year mortality among those with the highest LDL-C in the included cohorts was up to 36% lower than among those with the lowest LDL-C. Furthermore, in the largest study20 that included about two-thirds of the total number of participants in our study, the risk was lower among those with the highest LDL-C than among those on statin treatment.

It is also possible that those with the highest LDL-C were put on a different diet than those with low LDL-C. However, this potential bias in mortality outcomes could have gone in both directions. Some of the individuals with high LDL-C may have followed the official dietary guidelines and exchanged saturated fat with vegetable oils rich in linoleic acid. In a recent study, the authors reported that among participants who were older than 65 at baseline, a 30 mg/dL decrease in serum cholesterol was associated with a higher risk of death (HR 1.35, 95% CI 1.18 to 1.54).26 If applied to the general population, this finding suggests that the conventional dietary treatment for high cholesterol with vegetable oil replacing saturated fat may actually increase mortality in those individuals with high LDL-C. Thus, the lack of an association between LDL-C and mortality may have been even stronger than reported since the dietary intervention may have been counterproductive.

Finally, it is potentially relevant that we limited our literature search to PubMed. In preliminary searches with PubMed, OVID and EMBASE, we identified 17 relevant studies in PubMed, but only 2 in OVID and EMBASE, and these 2 studies were found in PubMed as well. Therefore, it is highly unlikely that there are studies with findings with divergent results from those we have reported here, as all of them reported either no association or an inverse association between LDL-C and mortality.
Discussion

Assessments of the association between serum cholesterol and mortality have been studied for decades, and extensive research has shown a weak association between total cholesterol and mortality in the elderly; several studies have even shown an inverse association. It is therefore surprising that there is an absence of a review of the literature on mortality and levels of LDL-C, which is routinely referred to as a causal agent in producing CVD4 and is a target of pharmacological treatment of CVD.

Our literature review has revealed either a lack of an association or an inverse association between LDL-C and mortality among people older than 60 years. In almost 80% of the total number of individuals, LDL-C was inversely associated with all-cause mortality and with statistical significance.

These findings provide a paradoxical contradiction to the cholesterol hypothesis. As atherosclerosis starts mainly in middle-aged people and becomes more pronounced with increasing age, the cholesterol hypothesis would predict that there should be a cumulative atherosclerotic burden, which would be expressed as greater CVD and all-cause mortality, in elderly people with high LDL-C levels.

Our results raise several relevant questions for future research. Why is high TC a risk factor for CVD in the young and middle-aged, but not in elderly people? Why does a subset of elderly people with high LDL-C live longer than people with low LDL-C? If high LDL-C is potentially beneficial for the elderly, then why does cholesterol-lowering treatment lower the risk of cardiovascular mortality? In the following we have tried to address some of these questions.
Inverse causation

A common argument to explain why low lipid values are associated with an increased mortality is inverse causation, meaning that serious diseases cause low cholesterol. However, this is not a likely explanation, because in five of the studies in table 1 terminal disease and mortality during the first years of observation were excluded. In spite of that, three of them showed that the highest mortality was seen among those with the lowest initial LDL-C with statistical significance.18 ,20 ,24
Is high LDL-C beneficial?

One hypothesis to address the inverse association between LDL-C and mortality is that low LDL-C increases susceptibility to fatal diseases. Support for this hypothesis is provided by animal and laboratory experiments from more than a dozen research groups which have shown that LDL binds to and inactivates a broad range of microorganisms and their toxic products.27 Diseases caused or aggravated by microorganisms may therefore occur more often in people with low cholesterol, as observed in many studies.28 In a meta-analysis of 19 cohort studies, for instance, performed by the National Heart, Lung and Blood Institute and including 68 406 deaths, TC was inversely associated with mortality from respiratory and gastrointestinal diseases, most of which are of an infectious origin.29 It is unlikely that these diseases caused the low TC, because the associations remained after the exclusion of deaths occurring during the first 5 years. In a study by Iribarren et al, more than 100 000 healthy individuals were followed for 15 years. At follow-up, those whose initial cholesterol level was lowest at the start had been hospitalised significantly more often because of an infectious disease that occurred later during the 15-year follow-up period.30 This study provides strong evidence that low cholesterol, recorded at a time when these people were healthy, could not have been caused by a disease they had not yet encountered.

Another explanation for an inverse association between LDL-C and mortality is that high cholesterol, and therefore high LDL-C, may protect against cancer. The reason may be that many cancer types are caused by viruses.31 Nine cohort studies including more than 140 000 individuals followed for 10–30 years have found an inverse association between cancer and TC measured at the start of the study, even after excluding deaths that occurred during the first 4 years.32 Furthermore, cholesterol-lowering experiments on rodents have resulted in cancer,33 and in several case–control studies of patients with cancer and controls matched for age and sex, significantly more patients with cancer have been on cholesterol-lowering treatment.32 In agreement with these findings, cancer mortality is significantly lower in individuals with familial hypercholesterolaemia.34

That high LDL-C may be protective is in accordance with the finding that LDL-C is lower than normal in patients with acute myocardial infarction. This has been documented repeatedly without a reasonable explanation.35–37 In one of the studies,37 the authors concluded that LDL-C evidently should be lowered even more, but at a follow-up 3 years later mortality was twice as high among those whose LDL-C had been lowered the most compared with those whose cholesterol was unchanged or lowered only a little. If high LDL-C were the cause, the effect should have been the opposite.
Conclusions

Our review provides the first comprehensive analysis of the literature about the association between LDL-C and mortality in the elderly. Since the main goal of prevention of disease is prolongation of life, all-cause mortality is the most important outcome, and is also the most easily defined outcome and least subject to bias. The cholesterol hypothesis predicts that LDL-C will be associated with increased all-cause and CV mortality. Our review has shown either a lack of an association or an inverse association between LDL-C and both all-cause and CV mortality. The cholesterol hypothesis seems to be in conflict with most of Bradford Hill’s criteria for causation, because of its lack of consistency, biological gradient and coherence. Our review provides the basis for more research about the cause of atherosclerosis and CVD and also for a re-evaluation of the guidelines for cardiovascular prevention, in particular because the benefits from statin treatment have been exaggerated.38–40
Acknowledgments

The study has been supported by a grant from Western Vascular Institute.
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They Are Killing Us From the Skies: Death by Respiratory Disease Has Skyrocketed



“Chemtrail Cough” is Sweeping the Nation — Death by Respiratory Disease Has Skyrocketed
February 11, 2018
http://www.unseen-pedia.com/chemtrail-cough-sweeping-nation-death-respiratory-disease-skyrocketed/

Have you noticed that everyone around you is congested and coughing? I am calling this new syndrome “chemtrail cough.”

My wife is coughing, I am coughing and now even our dogs are coughing. In just four years death by respiratory disease has skyrocketed from 8th in the world to 3rd and possibly even 2nd.

If someone in congress receives a letter with a little bit of white powder in it the capitol is evacuated and the media freaks out and starts screaming “terrorists.”

However, right over their heads, hundreds of tons of nano aluminum and barium are being sprayed on them daily and yet nobody seems to care.

Yesterday I heard Alex Jones start his broadcast by saying his throat hurt and his voice was messed up because of something in the air but he didn’t know what it was.

Just one hour before his show started I heard the world’s leading expert on the subject of chemtrailsand geoengineering (Dane Wiginton) conduct an interview and he sounded terrible too.

Alex often talks about what people will put up with. One of his ideas is to go door to door in Austin and ask homeowners if it would be OK to put cameras in their bedrooms and then record their responses.

Of course, no sane person would allow that yet they do allow the NSA and countless other agencies and private corporations to do exactly the same thing via their smart TVs and smart phones.

Here’s my idea. How would anyone from Infowars or any other patriot radio show like it if I cornered your kid somewhere, opened a can of bug spray and emptied the entire can on their face?

You probably wouldn’t like it too much and yet you tolerate the EXACT same thing to be done to your kids on a daily basis by Bill Gates and the rest of the geo-engineering fanatics.

How is this not the number one story in the world all day and every day? Has everyone in the alternative media lost their minds too? We know that nano sized particulate of aluminum is very harmful to the brain and is responsible for killing everything that lives and yet it is rarely discussed and very little effort is directed to exposing it and stopping it.

Don’t people understand that if we don’t stop this right now there will be nothing left to live and fight for?

They are turning the sky over the entire country white daily. I have saved these recent satellite images to illustrate just how much material they are dumping on us. The sprayers repeat the same pattern day after day with very few exceptions.

Satellite Images

Sunday morning (5/25/14)

Sunday afternoon (5/25/14)

Monday morning (5/26/14)

Monday afternoon

Tuesday morning (5/27/14)

Tuesday afternoon (5/27/14)

They typically spray heavily first thing in the morning and greet the sun as it rises. They then spray all day long following the sun as it crosses the sky. They then spray heavily in the west as the sun sets.

The aluminum and barium (blanketing us all day) dissipates and falls to the ground in the early evening. The planes then return to their bases to refuel, rearm and get ready for the next attack the next morning.

Unless all radio talk show hosts come together and put an end to this craziness called geo-engineering it is my opinion that all life on earth will die and the battle for the future of humanity will be lost.

Dane Wigington should be on every radio show at least once a week to cover the latest developments in the collapse of our ecosystem. It’s accelerating and nothing is being done to curtail these insane programs.

Source humansarefree